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Mourdikoudis, S., Montes-Garca, V., Rodal-Cedeira, S., Winckelmans, N., Prez-Juste, I., Wu, H., & Pastoriza-Santos, I

2019;doi:10.1136/esmoopen-2019-000540

In endometriosis, impaired endometrial receptivity arises from interconnected mechanisms involving defective decidualization, altered immune cell populations, and a disrupted endometrial environment, collectively contributing to implantation failure

Of note, 4-hydroxynonenal (4-HNE), as the end-product of lipid peroxidation, could increase the expression of Fas in a concentration- and time-dependent manner, accompanied by the activation of caspase 3 and finally leading to apoptosis [83]