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Increased apoptotic sensitivity of glioblastoma enables therapeutic targeting by BH3-mimetics

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In neuronal preparations, GCPRs activated by caffeine, glutamate via mGluR, GABA via GABA B receptors, or calcitonin-gene-related protein (CGRP) receptor via CGRP have been shown to induce release of BDNF (Balkowiec and Katz 2002

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