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Evidence suggests that oxidative stress from immune cells, orbital fibroblasts, and adipocytes infiltrating the orbit contributes to TAO pathogenesis (42)

Methods: We crossed Cflar FF ;Alb-Cre mice with Fgf18 FF mice, resulting in the generation of hepatocyte-specific Cflar and Fgf18 double-deficient ( Cflar LKO ;Fgf18 LKO ) mice