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Metabolic response in the late 72 h reperfusion time suggests that neural cells accelerate glutamate metabolic recovery to substitute the deprivation induced by ischemic event and presumably compensate for the altered synthetic function of damaged neural cells

Hussain A, Saikia V, Ramteke A (2012) Nitric oxide and modulatory effects of the root extracts of Phlogacanthus tubiflorus against oxidative stress induced by hydrogen peroxide

Methods: Data for DRFs was obtained from the Organ Procurement & Transplantation Network (OPTN) website via the US Department of Health & Human Services, and the Health Resources & Services Administration (HRSA), and was cross-referenced using Google Maps and direct contact with organ procurement organization staff

Dysfunction in mitochondria can lead to electron leakage in the electron transport chain, resulting in overproduction of ROS such as superoxide anions and hydrogen peroxide ( Endoplasmic reticulum oxidative stress: The endoplasmic reticulum (ER), responsible for protein synthesis and folding, is vulnerable to oxidative stress