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Increased binding was present in the hippocampus, amygdala, thalamus, midbrain, pons, and cingulate cortex, although the exact area of the cingulate cortex was not defined

10.1016/j.ejca.2019.05.008 314 RedmanB

infusion of glutathione or its precursor, N-acetyl cysteine (NAC).

However, these models fail to explain how disease-specific monomeric Syn variants orchestrate the sequestration of diverse cellular proteins and damaged membrane organelles to initiate and promote LB formation