glutathione breast cancer metastasis

glutathione breast cancer metastasis Neutrophils resist ferroptosis and promote through aconitate decarboxylase 1: Cell Metabolism MitoWorld

Name: glutathione breast cancer metastasis Neutrophils resist ferroptosis and promote through aconitate decarboxylase 1: Cell Metabolism MitoWorld
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SKU: 38328394351
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MitoWorld Metabolic Reprogramming Encyclopedia MDPI Glutathione Peroxidases: An Emerging and Promising Therapeutic Target for Pancreatic Cancer Treatment GPX4 Inhibitor Resistance and Metastatic Features in TripleNegative Breast Cancer Sabatier 2026 Advanced Science Wiley Online Library

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The C-terminal domain induces the production of myeloperoxidase, TNF-, and tissue factor, while the N-terminal domain (miniYRS) interacts with CXCR1/2 receptors to influence the migration of polymorphonuclear cells (PMN), promoting monomer formation and inhibiting the dimer formation (Vo et al., 2011)