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Amniotic MSCs-derived EXOs promote trophoblast proliferation by blocking the EZH2/mTOR signaling pathway, which in turn enhances autophagy in trophoblasts ( Even though EXOs have much promise for therapeutic uses, their restricted capacity to target in animal trials results in issues such as short half-lives and decreased therapeutic effectiveness

Mammalian soluble epoxide hydrolase is identical to liver hepoxilin hydrolase

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This effect was accompanied by a significant reduction in released cytosolic proteins such as myoglobin and creatine kinase [50, 52, 53] as well as attenuation in plasma marker of oxidative stress - malondialdehyde [51, 53, 54]