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& Shen, J

NNMT inhibitors show promise in suppressing obesity and increasing metabolic health

Metabolic reprogramming of macrophages: glucose transporter 1 (GLUT1)-Mediated glucose metabolism drives a proinflammatory phenotype

Thus, radiation-induced Drp1-mediated mitochondrial fission serves as a critical pro-survival mechanism that inhibits apoptosis, promotes mitophagy and metabolic reprogramming, and drives radioresistance, whereas blocking fission shifts the balance toward mitochondrial elongation, cell cycle arrest, and enhanced radiation sensitivity -underscoring Drp1 as a compelling therapeutic target to overcome radioresistance