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It primarily manifests in the intima of affected arteries as lipid depositions, infiltrations of monocytes and lymphocytes, the migration and proliferation of vascular smooth muscle cells (VSMCs), and the formation of foam cells, lipid striations, and fibrous plaques, which further contributes to vascular wall sclerosis, stenosis, and thrombosis

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2021;162, Calderon-Dominguez M, Sebastin D, Fucho R, Weber M, Mir JF, Garca-Casarrubios E, et al