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at the same time, it can maintain the stable expression of estrogen receptor (ER) and (ER), and activation of either receptor can induce protective autophagy, while also inhibiting the mTOR pathway to form a synergistic effect, thereby upregulating insulin signaling to protect islet cells and inhibit their apoptosis ( in vitro and in vivo experiments have found that silybin improves fatty liver and insulin resistance primarily by targeting and downregulating the expression of the microRNA miR-122