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Receptor tyrosine kinase-dependent Pi3k activation is an escape mechanism to vertical suppression of the Egfr/Ras/Mapk pathway in kras-mutated human colorectal cancer cell lines

The treatments used are given below: 0 mM (control) 100 mM Na 2 CO 3 150 mM Na 2 CO 3 0 mM (control) + JA 100 mM Na 2 CO 3 + JA 150 mM Na 2 CO 3 + JA Estimation of plant growth, biomass yield, and total chlorophyll The root and shoot lengths were measured using a manual scale

Given the small sample size, additional covariate adjustment was not performed to avoid model overfitting, and residual age effects cannot be fully excluded

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