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The study supports the concept that GHSs may provide a safer and more physiologic alternative to exogenous GH therapy by stimulating endogenous pulsatile GH release, thereby avoiding supraphysiologic IGF-1 levels [2]

PD is characterised by a progressive loss of, primarily, dopaminergic neurons of the substantia nigra that project to the striatum, resulting in its motor symptoms of bradykinesia, rigidity and tremor

However, after aSAH, various oxidative cascades, driven by increased metabolic requirements and compromised cellular respiration, overwhelm these intrinsic antioxidant defenses, leading to further damage

Urolithins: a comprehensive update on their metabolism, bioactivity, and associated gut microbiota