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Researchers at Monash University in Australia first isolated the fragment in the late 1990s, hypothesizing it could reproduce the lipolytic (fat-burning) effects of growth hormone without triggering the diabetogenic and growth-promoting side effects [1]

In short: The mechanism makes sense The animal data is compelling The human evidence is incomplete Anyone approaching BPC-157 should do so with informed caution, realistic expectations, and respect for the current limits of science

One key difference is that keloid scars are found extending outwards out of the normal tissue boundary, while hypertrophic scars are confined in the boundaries

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