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Description

Anti-inflammatory effects of BHBA in both in vivo and in vitro Parkinsons disease models are mediated by GPR109A-dependent mechanisms

10.1016/S0304-3940(98)00129-3 80 SimpsonI

While GHK has been shown to inhibit the inflammatory response and epithelial-mesenchymal transition (EMT) via the TGF-1/Smad 2/3 and IGF-1 pathways in bleomycin-induced fibrosis models [24], its broader anti-inflammatory effects in the context of pulmonary fibrosis pathogenesis may warrant further exploration

Furthermore, leucine, glutamate and glutamine remarkably influence the atherogenicity of macrophages through modulating cellular triglyceride metabolism (167)