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Loss of autophagy in erythroid cells leads to defective removal of mitochondria and severe anemia in vivo

doi: 10.1083/jcb.201202014

Currently, transgenic mouse models overexpressing A53T and A30P mutations are extensively utilized

However, the effects of different exercise types on mitophagy and the potential mechanism by which exercise regulates mitophagy to intervene in metabolic diseases remains unclear