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Biomacromolecules 16 , 798806 (2015)

This review systematically elucidates core pathways through which MPs/NPs drive PD pathology via multidimensional molecular mechanisms: (1) MPs/NPs facilitate -synuclein oligomerization and fibrillation by hydrophobically adsorbing its NAC domain

The mechanism of treatment employs using the intrinsic cellular energy to reduce the active agent lipoate-[S-S] (actually a pro-drug) to dihydrolipoate [DHLA-(SH) 2 ] (the reduced active agent)

These data are in line with the previous study reported in vitro for macrophages and human smooth muscle cells, in which -tocopherol inhibits uptake of oxLDL by downregulating CD36 expression (Devaraj et al., 2001)