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OT-82 is a promising antineoplastic agent for the study of hematological malignancies

doi: 10.1146/annurev-arplant-081320-092855

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Fasn inhibition was shown to reduce endothelial damage and functional changes by suppressing oxidative stress and NLRP3 inflammasome-dependent pyroptosis through activation of the Nrf2/HO-1 pathway