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In the acute phase of AD, Th2-mediated immune reactions initiate the release of pro-inflammatory cytokines, further perpetuating the inflammatory cascade (45)

This imbalance contributes to mitochondrial fragmentation, functional impairment, and oxidative stress, all of which exacerbate endothelial cell dysfunction, apoptosis, and impaired wound healing (Sun et al., 2016)

Signature of Al11Sm3 fragments in undercooled Al90Sm10 liquid from ab initio molecular dynamics simulations

Both chemotherapeutic agents also lead to increased levels of proinflammatory cytokines (IL-6, IL-8, IL1, TNF-), which can lead to sensitization of nociceptors (Ledeboer et al., 2007