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This anti-inflammatory effect was mechanistically linked to niacins ability to attenuate oxidative stress by inhibiting NADPH oxidase activity and reducing ROS production, a key driver of inflammatory signaling in NAFLD pathogenesis ( However, the anti-inflammatory role of VB3 is not universally observed across all experimental models, as evidenced by a controversial study by Further supporting a role for VB3, recent evidence continues to highlight the therapeutic potential of nicotinamide

Consistent with the in vitro data that the co-treatment of R001 with NAC blocks R001-induced ROS production (Fig

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Remarkably, the elimination of 7nAChR nullified SINs inhibition of NF-B activation, indicating a reliance on 7nAChR for its anti-inflammatory effects