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Activation of c-Met by HGF leads to a variety of cellular signals that mediate tumor growth, metastasis, and angiogenesis

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Accumulation of A aggregates and longer As in Rab7-positive late endosomes and cathepsin D-positive autophagolysosome and mitochondria suggest that age-related changes in iA processing limit completion of autophagic and endocytic processes in aged neurons [1]

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