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Interestingly, activation of mGlu3 receptors with the endogenous agonist, N-acetylaspartylglutamate (NAAG), enhances delay in cell firing in the dorsolateral PFC during a working memory task, improving cognitive function 174

Potentiation of nicotinic receptor response by external calcium in rat central neurons

Further, the glutathione S-transferase (GST) enzyme conjugates GSH to harmful compounds, such as oxidized lipids, converting them into less toxic and more easily excreted substances [16]