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Activation of UCP1, a proton channel in the inner mitochondrial membrane of brown adipocytes, produces a leak in the mitochondrial membrane and uncouples ATP synthesis from respiration, thereby dissipating energy as heat (6, 100)

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It is prepared in accordance with Section 503A of the Federal Food, Drug, and Cosmetic Act and is dispensed solely pursuant to a valid patient-specific prescription from a licensed healthcare provider

Researchers have proposed that AOD 9604 may operate by potentially eliciting fat breakdown while impeding the formation of new fat stores in laboratory settings