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Deficient proteostasis allows the transition of amyloid fibrils from soluble, amorphous states into insoluble, crystalline -sheet aggregates [185], via breaching solubility limits coupled with a breakdown of supersaturationpromoted by defective clearancewhere the solute concentration overwhelms its thermodynamic solubility [186]

It is hoped that the results of this exploratory study will help to promote further research on the clinical significance of hypocarnitinemia in patients with eating disorders and the need for carnitine replacement therapy

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