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Understanding how GSH level and synthesis velocity depend on the sex hormones is an extremely important question since oxidative stress contributes to the risk for heart disease [8] and cancer [9] , and oxidative stress is reduced by GSH via the enzyme GPX [Figure 1]

However, upon inhibition of the System XC-, cysteine is denied cellular entry, necessitating an alternative cysteine-dependent mechanism to resist ferroptosis (Tang and Kang 2023)

These studies suggested that the antidepressant-like effect of PF or TGP may be realized by protecting monoamine neurotransmitters

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