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Peptidase therapy also reduced the infiltration of the pancreas by MPO neutrophils and F4/80 monocytes (120), we believe these data support future CXCR2-based therapeutic interventions

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Boosting energy supplies produces excess free radicals from increased oxidative phosphorylation [14] which may not be neutralized if the cells have been operating in a low redox capacity with low activation of Antioxidant Response Elements (ARE) genes

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