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Figure 5 4 Promising therapeutical reagents for SLE by inhibiting ferroptosis Given that several pathophysiological features of ferroptosis, including iron accumulation, mitochondrial dysfunction, overproduction of lipid peroxidation and suppression xCT-GPX4 pathway are potentially implicated in SLE progression, reversing those features may be beneficial to SLE disease

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In our study, we demonstrated a persistent senescent-inflammatory microenvironment and a p53-serine 15 phosphorylation regulatory mechanism in keloids, which may play crucial roles in the aggression and recurrence of keloids