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Studies in TLR9-activated pDCs have shown that they require enhanced FAO and OXPHOS via an autocrine IFN-I receptor-dependent pathway to support robust IFN-I production under physiological conditions ( Furthermore, the role of key glycolytic enzymes in DCs may differ from that observed in other immune cells

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Researchers combine them to probe whether parallel pathway engagement produces additive or distinct effects in connective-tissue, gastrointestinal and cardiovascular injury models