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Kwizera EN, Hulbert PB and Hicks R

Furthermore, we identified the underlying molecular mechanisms: Wnt induction of IGF1 depends on -catenin and subsequent binding of Wnt transcription factor TCF4 to the IGF1 gene promoter

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At the same time, these mediators drive heightened mitochondrial respiration in chondrocytes, generating excess reactive oxygen species (ROS) that damage mitochondria and promote chondrocyte apoptosis and autophagy (Vonk et al., 2018