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Although many studies have shown that GDNF can promote longdistance regeneration of axons after spinal cord injuries, the uncontrolled expression of GDNF may have side effects owing to an excessive axonal growth, aberrant sprouting, and axonal entrapment resulting in a pathological hypertrophy of the injured nerve (Eggers et al., 2019)

While less structurally complex than tendon or labral injuries, bursitis can prove stubbornly persistent when the underlying cause remains unaddressed

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