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The hypothalamic-pituitary-adrenal (HPA) axis plays a central role in the stress response, and prolonged adverse stress may contribute to HPA axis dysregulation, including altered cortisol rhythmicity and impaired negative feedback regulation

Incomplete or insufficient response: Failure to achieve remission characterized by the lack of normalization of transaminases and immunoglobulin G levels within 6 months despite adherence to therapy

Responses vary with concentration and exposure time (biphasic effects are described in some settings), the proinflammatory stimulus (e.g., LPS vs

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