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Experimental observations suggest that the mitigation of microRNA 146a expression may reverse the suppressive implications of thymosin beta 4 on IRAK1 and TRAF6 signaling activity

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both peptides are studied in animal models of tendon, ligament and muscle healing

Perspectives and challenges Abnormal activation of Wnt/-catenin signalling promotes the progression of various diseases and oncogenic transformation of tumours, making key factors involved in Wnt/-catenin signal transduction attractive therapeutic targets