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(2017) 356:j831

Indeed, in vivo research has demonstrated downregulation of fetal brain drug transporters in term IUGR, suggesting increased brain-specific bioavailability that may explain the adverse neurodevelopmental outcomes observed in IUGR subjects exposed to ACS (285)

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Alternatively, the more supported hypothesis is that there could be hyper-responsivity of the cholinergic receptors at a hypothalamic level, causing a greater decrease in somatostatin tone, and elevated GH release from the anterior pituitary gland (153)