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A previous study reported that vitamin C could prevent monosodium iodoacetate (MIA) induced changes such as cell growth inhibiting and oxidative stress increasing, apoptosis, MMPs increasing, and proteoglycan loss in a chondrosarcoma cell line (SW1353) [160]

These measures obviously should not be considered as challengers of pharmacological treatments in very high-risk individuals in primary prevention but more as partners, as both provide complementary benefits by decreasing major cardiovascular events and mortality.

This work is supported, in part, by National Institutes of Health grants R01CA291876, R01CA284108, R01NS117828 and T32-CA196561-08, American Cancer Society grant RSG-22-052-01-IBCD, Cancer Prevention and Research Institute of Texas grants RP240493 and RP250191, the American Brain Tumor Association, the Radiological Society of North America, the Red Gates Foundation and Colorectal Cancer Alliance