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Increased blood glucose levels induce excessive mitochondrial fission or fragmentation, thereby affecting OXPHOS, inhibit the expression of PINK1 and Parkin and downregulate the expression of genes such as PGC-1, thereby inhibiting mitochondrial biogenesis and mitophagy and preventing cells from eliminating abnormal mitochondria

Levels naturally decline with age, which has fuelled interest in whether supplementation can reverse age-related tissue changes

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Also, while writers are shared, the specific eraser machinery (delactylases) remains less characterized than HDACs, representing a critical gap in the field