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Retrieved from Sharabi, A., Cohen, E., Sulkes, J., & Garty, M

[57] AD is associated with an accumulation of DNA damage (double-strand breaks) in vulnerable neuronal and glial cell populations from early stages onward, [58] and DNA double-strand breaks are increased in the hippocampus of AD brains compared to non-AD control brains

[1] Research suggests these findings might indicate that BPC157 supports tendon fibroblast signaling primarily through post-translational modulation of FAK-paxillin-associated pathways governing F-actin assembly, cellular adhesion, and directional migration

10.3389/fimmu.2018.00784 220 YaribeygiH.SathyapalanT.AtkinS