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We found that Gstp / mice displayed a higher mortality rate, severer acute lung injury and higher HMGB1 release level than wild type C57BL/6 mice after CLP surgery (Figures 1AC), which first demonstrated that GSTP acted as a key negative regulator in sepsis development

Given that BNIP3 has been observed to heterodimerise with BCL-2 in vitro 12 , we examined the response to B-017 in staurosporine-treated MEFs lacking BCL-2 family members in terms of late-stage cell death signalling (Fig

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