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Description

TPP-CeO 2 exhibits excellent long-term biocompatibility within skin tissues, showing minimal cytotoxicity and no significant structural or functional damage to the skin, thereby supporting its safety for treating chronic inflammatory conditions such as psoriasis

Dysbiosis may also disrupt the metabolome, further compromising the mucosal barrier and contributing to inflammation

The mutant fragments were cloned into pK18, a suicide vector, to generate gene knockout plasmids

Pro-inflammatory cytokines such as TNF- , IL-1, and IL-6 can enhance BBB permeability by altering tight junction proteins, resulting to a loss of barrier integrity