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Dissociation constants for the interaction between ZZ with unlabeled peptides H3 (112) and H3K4ac (112) were measured using a displacement assay in which increasing amounts of unlabeled peptides were added into a preformed ZZ:H3-FAM complex prepared by supplementing 5 M ZZ into each sample

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Amyloid-beta oligomers, which are precursors to amyloid plaques in the Alzheimers disease brain, stimulate mTORC1 kinase activity at the plasma membrane but not at lysosomes and block nutrient-induced mitochondrial activity (NiMA) through a mechanism dependent on tau