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Although these disorders are very rare, they underscore the fundamental link between disrupted glutathione homeostasis and pyroglutamate accumulation [75]

10.1016/j.seizure.2008.11.006 7

Compared to the non-CV death group, the CV death group included more females (60.0% vs 23.1%, p=0.049) and recipients with a prior diagnosis of atrial fibrillation pre-LT (40.0% vs 10.3%, p=0.044)

A likely possibility is that homoglutathione replaces glutathione as the dominant cellular redox-buffer in tissues associated with nitrogen fixation, as homoglutathione is required for proper development of nodules (Frendo et al., 2005)