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2 O predominantly affect glutamatergic transmission by antagonizing NMDA receptors (NMDARs), causing excitotoxicity, mitochondrial dysfunction, and neuronal apoptosis

The combinational effects of N-terminal mutations and C-terminal affinity-enhancing mutations shown in this study remain to be tested for the future study

This indicates the efficacy of using MET inhibitors alongside ICIs in treating tumors where MET is dysregulated and PD-L1 expression is elevated