glutathione s-transferase monoclonal antibody s transferase theta 2 Sigma-Aldrich Anti- Glutathione-S-Transferase (GST) produced in mouse, clone GST-2, ascites fluid 100 uL P and reacts with Human Glutathione pi 1/GSTP1 Antibody – linked immunosorbent assay (ELISA) Anti Glutathione S-transferase Mu 5 Antibody

Heterogeneity was assessed using the I 2 statistic, which describes the proportion of variation in the log odds ratios that is attributable to genuine differences across studies rather than to random error [150], and by the 2 test [151]

However, these criteria are difficult to use in AML because these patients are prone to bacterial and fungal infections [184,185,186], have reduced levels of circulating normal cells due to the disease or the chemotherapy [184,185,186,187], the disease itself may cause splenomegaly [188,189], fibrinogen may be increased due to an acute-phase reaction [190] (see Section 5.2), they often receive nutritional support that makes it difficult to interpret the triglyceride level [191], hemophagocytosis may be seen without HLH [192] and increased ferritin levels may have other causes including an iron overload

Minimal inhibitory concentrations are on par or lower than gold standard medications, even among drug resistant isolates

doi: 10.1016/j.drudis.2008.11.006 14 BrancatoVOliveiraJMCorreloVMReisRLKunduSC