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Description

cited 2008 Apr

Magnsdttir, S., Ravcheev, D., De Crcy-Lagard, V

Oxidative damage extends beyond the acute phase, disrupting the BBB, impairing vascular integrity, and activating inflammatory cascades that perpetuate secondary injury

DNA-damaging agents, such as topoisomerase I inhibitors and DNA alkylators, induce replication stress, chromatin destabilization, and double-strand breaks, culminating in the activation of DNA damage checkpoints, G2/M arrest, and apoptotic cell death [24, 123]