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Livak and Schmittgen 2001)

Loss of GPX4 protein expression in NRF1-deficient cells sensitized cells to ferroptosis, whereas in cells lacking GPX4, overexpression of NRF1 failed to inhibit ferroptosis

Selenium-dependent enzymes in the dogs body convert T4 (the mostly inactive thyroid prohormone) into T3 (the active form that cells use)

These anti-inflammatory and antioxidant effects translate into histological protection (less neuronal loss) and functional improvements in motor behavior